Graduated at the medical school from Universidade Federal do Espírito Santo (2004) and fellow in Internal Medicine (2007) and rheumatology (2009) at Universidade Federal de São Paulo. PhD in rheumatology at Universidade Federal de São Paulo. Postodoctoral at the University of Washington and fellowship in Seattle Children's Research Institute/Center for immunity and immunotherapies. Currently holds the following positions: Affiliated Professor and chief of the Immune dysregulation Clinics of the Rheumatology Division at Universidade Federal de São Paulo; Physician of the Immunology Division of the Central Laboratory at Hospital das Clínicas da Faculdade de Medicina da Universidade de São Paulo; Dean of the Rare Diseases Commitee of the Brazilian Rheumatology Society; Member of the Midia Commitee of the Paulista Rheumatology Society. Has experience in Medicine, focusing on Internal Medicine and Rheumatology, acting on the following subjects: immune dysregulation, primary immune regulatory diseases, autoinflammatory diseases, autoimmune rheumatic diseases, systemic lupus erythematosus, Behçet´s disease, immunodeficiency, immunology, rheumatoid arthritis. (Source: Lattes Curriculum)
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Behçet's Disease (BD) is currently classified as a polygenic autoinflammatory syndrome. Recently point mutations in NFKB1, TNFAIP3 and NEMO were associated with BD-like phenotypes, showing the importance of the NF-kB pathway on BD pathogenesis. Constitutional or acquired trisomy 8, which encodes IKBKB, an important NF-kB activator, is often associated with BD. In addition, sCD40L, which, …
Behçet's disease (BD) is currently classified as a polygenic autoinflammatory syndrome. Recently point mutations in NFKB1, TNFAIP3 and NEMO were associated with BD-like phenotypes, showing the importance of the NF-kB pathway on BD pathogenesis. Constitutional or acquired trisomy 8, which encodes IKBKB, an important NF-kB activator, is often associated with BD. In addition, sCD40L, which, …
Behçet's disease (BD) is currently classified as a polygenic autoinflammatory syndrome. Recently point mutations in NFKB1, TNFAIP3 and NEMO were associated with BD-like phenotypes, showing the importance of the NF-kB pathway on BD pathogenesis. Constitutional or acquired trisomy 8, which encodes IKBKB, an important NF-KB activator, is often associated with BD. In addition, sCD40L, which, …
Introduction: Systemic Lupus Erythematosus (SLE) is one of the most common systemic auto-immune disease and considered the prototype for systemic auto-immune condition. The beginning of the illness and its variation in intensity result of innumerable factors: hormones, microrganisms, UV light, stress, diet, genetics, amongst others. In all the cases, there seems to be the participation o…
| 1 / 1 | Ongoing research grants |
| 3 / 3 | Completed scholarships in Brazil |
| 4 / 4 | All research grants and scholarships |
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CV Lattes
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