Abstract
One of the aims of the CRID/CEPID project involves the characterization of the connection of endoplasmic reticulum (ER) stress, autophagy, inflammasome and nitric oxide production. We will evaluate the effect of the autophagy and ER stress separately into each process. This will be achieved by using gene knockdown and Atg5flox/flox/Lysm-Cre mice for autophagy evaluation during Leishmania …