Abstract
Renal ischemia/reperfusion injury (I/R)is the main cause of intrinsic ARF in the native and transplant kidneys. Recent data demonstrated that T CD4+ cells play an essential role as a mediator of kidneyaggression in I/R. However, the effector mechanisms involved are still poor understand, what complicates the drawing of strategies for limit the tissue injury. In this project, we hypothesiz…