CV Lattes
Universidade de São Paulo (USP). Faculdade de Medicina de Ribeirão Preto (FMRP) (Institutional affiliation from the last research proposal) Birthplace: Brazil
I graduated at Biological Sciences from University of the State of São Paulo- UNESP (2005), then I've accomplished my master's degree (2010) and Ph.D. degree (2014) in Cell and Molecular Biology and a post-doctorate in Genetics (2017) at the Ribeirão Preto Medical School - University of São Paulo (USP). Part of my PhD was developed at CUMC in NYC (NY-USA). Also, I had been working at the Lab of Genetics and Bioinformatics on the Blood Center Foundation in Ribeirão Preto as a researcher fellow from Technological and Industrial Development Program from CNPq, collaborating with research activities involved in the molecular biology of solid tumors and the search for new targets for anti-cancer therapy. I am currently a Collaborating Professor at the State University of the Midwest (UNICENTRO), teaching in the areas of Morphology and Genetics for the Biological Sciences, Nursing, Pharmacy and Medicine courses; and a member of the Genomic Network of the Institute for Cancer Research (IPEC) in Guarapuava / PR. My expertise is on Cellular and Molecular Biology and Genetics, specially on the following themes: culturing of tumor cells, in vitro assays of cellular and molecular biology, confocal microscopy, analysis of gene expression, new generation sequencing, regulation by microRNAs and cancer. (Source: Lattes Curriculum)
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Myosins constitute a diverse superfamily of actin-based molecular motors that perform a wide range of fundamental functions, including cytokinesis, cell migration, cell contraction and the intracellular transport and correct positioning of vesicles, organelles and molecules. Recently, it was shown that myosin Va (MVa) is involved in cell migration and is up-regulated in metastatic tumor c…
Focal adhesion turnover is crucial for cell migration. Through this process, focal complexes disassemble and their components reassemble into new focal contacts. Focal adhesion disassembly occurs through clathrin-dependent endocytosis of integrin and also requires microtubule and FAK. Endocytosed integrins entering early endosomal vesicles (Rab5) end up eventually to late endosomes, but m…
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