CV Lattes
Universidade de São Paulo (USP). Faculdade de Medicina de Ribeirão Preto (FMRP) (Institutional affiliation from the last research proposal) Birthplace: Brazil
Silvia Cellone Trevelin is a member of European Society of Cardiology (ESC) and British Society for Immunology (BSI). She has experience in Pharmacology and Immunology with emphasis in sepsis, cardiovascular diseases, reactive oxygen species generated by NADPH oxidases and toll like receptors. She is graduated in Veterinary Medicine in 2008 at School of Veterinary Medicine and Animal Science (UNESP-SP, Brazil). In 2010, she undertook her master degree and doctoral research at Department of Pharmacology, Faculty of Medicine of Ribeirao Preto (FMRP-USP-SP, Brazil). She developed her PhD studies at Laboratory of Inflammation and Pain headed by Prof. Fernando de Queiroz Cunha (FMRP-USP). Her studies were also developed in collaboration with The Laboratory of Redox Signalling (Institute of Biological sciences- USP-SP) headed by Prof. Lucia Rossetti Lopes. In 2013, she also developed some experiments at Department of Cardiology, James Black Centre, King's College London, under supervision of Professor Ajay M. Shah. In other to develop her studies, she received financial support from European Society of Pharmacology (ESC), British Heart Foundation and Fundação de Amparo a Pesquisa do Estado de São Paulo (FAPESP). Currently, she is a research associate at King?s College London. (Source: Lattes Curriculum)
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Neutrophils are the first defense line of the organism to bacterial infections, and their recruitment and generation of reactive oxygen and nitrogen species (ROS and RNS) are essential to combat the microorganisms. In spite of the important microbicidal role, this substances may oxide cellular components and interfere with their function. The ability of nitric oxide and peroxinitrite to r…
Sepsis is a major cause of mortality in intensive care units. This syndrome develops when the host response to infection is inadequate resulting in bacteremia, systemic inflammation, cardiovascular collapse and death. Vascular dysfunction in sepsis may culminate in a state of shock characterized by prolonged hypotension, unresponsive to vasoconstrictor agents. This process results in mult…
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