Abstract
Myocardial infarction (MI) causes irreversible loss of cardiomyocytes with replacement by fibrous tissue, resulting in adverse cardiac remodeling and heart failure (HF). This is ideal for the development of complex ventricular arrhythmias and, consequently, ventricular tachycardia (VT) and fibrillation (VF) is routine in clinical practice. In recent years, considering the well-documented …